https://www.prisonplanet.com/did-you-kn ... oride.html
http://www.antioxidants-for-health-and- ... fects.htmlTo make matters much worse for human health, fluorides in teas are found together with aluminum. The combination of aluminum and fluorides in tea is of urgent concern, due to the increased damage done by fluorides when in the presence of aluminum, especially neurological and renal damage. It also increases the extent to which aluminum can be absorbed by the body, which has been linked to Alzheimer’s disease.
Aluminum by itself is not readily absorbed by the body, however in the presence of fluoride ions, the fluoride ions combine with the aluminum to form aluminum fluoride, which is absorbed by the body. Aluminum eventually combines with oxygen to form aluminum oxide or alumina. Alumina is the compound of aluminum that is found in the brains of Alzheimer’s disease.
https://www.peoplespharmacy.com/2014/04 ... -my-brain/Green tea often contains high levels of aluminum. Excessive aluminum increases free radical production and leads to serious bone and brain disorders. There is a strong link between aluminum levels in the brain and neurodegenerative diseases such as Alzheimer's and Parkinson's disease.
“For many years aluminum has been considered innocuous for human beings, since most chemical forms are not damaging for living organisms. However, if pH soil values are low, aluminum tends to form chemical species that are potentially absorbed by plants, especially tea, and become toxic for living organisms. In this sense, there has been controversy on the impact of this metal on biological systems particularly in the last years. Concerning human health recent studies have demonstrated that bioavailable aluminum is related to some diseases such as Alzheimer, Parkinson, and dialysis encephalopathy.”
…Mechanisms that underlie the risk of low concentrations of aluminum relate to (1) aluminum’s absorption rates, allowing the impression that aluminum is safe to ingest and as an additive in food and drinking water treatment, (2) aluminum’s slow progressive uptake into the brain over a long prodromal phase, and (3) aluminum’s similarity to iron, in terms of ionic size, allows aluminum to use iron-evolved mechanisms to enter the highly-active, iron-dependent cells responsible for memory processing. Aluminum particularly accumulates in these iron-dependent cells to toxic levels, dysregulating iron homeostasis and causing microtubule depletion, eventually producing changes that result in disconnection of neuronal afferents and efferents, loss of function and regional atrophy consistent with MRI findings in AD [Alzheimer’s disease] brains. AD is a human form of chronic aluminum neurotoxicity. The causality analysis demonstrates that chronic aluminum intake causes AD.